Objective: To investigate the impact of low intensity isocapnic hyperpnoea (IH) on blood lactate disappearance after exhaustive arm exercise in comparison to passive and active recovery using the previously loaded muscle group.
Design: Randomised, crossover.
Setting: Institute for Sports Medicine.
Participants: 18 healthy non-smoking and physically active male subjects.
Interventions: Subjects performed three arm cranking tests to volitional exhaustion on 3 different days at least 48h apart. Arm exercise was randomly followed by 30min of passive recovery (PR), active arm cranking at 30% of peak power output (AC) or ventilatory recovery (VR) by means of IH at 30% of 12s maximal voluntary ventilation. Blood lactate concentrations were measured every 2min during recovery.
Main Outcome Measurements: Blood lactate disappearance during the three different recovery strategies.
Results: No significant differences for blood lactate concentrations were found between interventions PR, AC and VR during the whole measurement period. Peak lactate concentrations were 11.09±1.98mmol•L-1 for PR, 11.13±1.44mmol•L-1 for AC and 11.25±1.93mmol•L-1 for VR. At the end of the recovery period measured lactate concentrations were 4.35±1.56mmol•L-1 for PR, 3.77±1.60mmol•L-1 for AC and 4.09±1.35mmol•L-1 for VR. Moreover, all other parameters meas-ured were not significantly different, with the exception of higher average recovery heart rates during AC (116±9bpm) and VR (111±7bpm) compared to PR (93±11bpm).
Conclusion: Low intensity IH seems not to enhance blood lactate disappearance after exhaustive arm exercise compared to passive or active recovery using the previously loaded muscle group. The magnitude of the involved muscle mass appears critical to effective active recovery.
- active recovery
- lactate kinetics
- respiratory muscles
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