TableĀ 3

Added value of CMR in the diagnosis and differentiation of cardiomyopathies

CardiomyopathyTypical pattern of fibrosis seen on CMR which allows differentiation from Athletes Heart
HCMClassically, fibrosis at the junction of the right ventricle and interventricular septum
Ischaemic DCMSubendocardial extending to transmural fibrosis, generally restricted to the perfusion territory of one coronary artery
Non-ischaemic DCMPatchy, mid-wall distribution in 28%.
Sub-endocardial pattern indistinguishable from ischaemic cardiomyopathy in 13%
ARVCDifferentiated from Athlete's Heart as RV and LV show disproportionate changes.
LVNCNon-compacted myocardium
Differentiated from Athlete's Heart as significant fibrosis in 55% of patients, which may occupy up to 5% of LV myocardium
MyocarditisMost commonly fibrosis has been shown to involve the epicardium of the inferior lateral wall.
Differentiated from Athlete's Heart due to lack of overt arrhythmias or classical symptoms (palpitations, presyncope or syncope)
  • CMR, cardiovascular magnetic resonance; HCM,hypertrophic cardiomyopathy; DCM, dilated cardiomyopathy; ARVC,arrhythmogenic right ventricular cardiomyopathy; LVNC,left ventricular non-compaction