Abstract
Prior exercise training protects skeletal muscle from contraction-induced injury. However, the mechanisms of protection are unknown. In this paper, the hypothesis is developed that exercise-induced increases in small heat shock protein activity may protect muscle cells via interactions with cytoskeletal elements and/or the glutathione system.
MeSH terms
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Animals
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Cytoprotection / physiology
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Exercise / physiology
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Glutathione / physiology
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Heat-Shock Proteins / physiology*
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Humans
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Muscle Contraction / physiology
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Muscle, Skeletal / injuries
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Muscle, Skeletal / metabolism
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Muscle, Skeletal / physiology*
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United States
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Wounds and Injuries / prevention & control*
Substances
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Heat-Shock Proteins
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Glutathione