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Correspondence
Inflammation and the continuum model: time to acknowledge the molecular era of tendinopathy
  1. Neal L Millar1,
  2. Benjamin J Dean2,
  3. Stephanie G Dakin2
  1. 1 Institute of Infection, Immunity and Inflammation, College of Medicine, Veterinary and Life Sciences, University of Glasgow, Glasgow, UK
  2. 2 Nuffield Department of Orthopaedics, Rheumatology and Musculoskeletal Sciences, Botnar Research Centre, University of Oxford, Oxford, UK
  1. Correspondence to Neal L Millar, Institute of Infection, Immunity and Inflammation, College of Medicine, Veterinary and Life Sciences, University of Glasgow, 120 University Avenue, Glasgow G12 8QQ, UK; neal.millar{at}glasgow.ac.uk

https://doi.org/10.1136/bjsports-2016-096419

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    The original continuum theory mentioned little concerning inflammatory mechanisms in tendon disease, likely due to the lack of scientific studies at that time. Since then, we and others have clearly defined a role for inflammatory cells1 and the subsequent inflammatory/matrix crosstalk involving cytokine regulation2 in human tendinopathy. It is …

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    Footnotes

    • Competing interests None declared.

    • Provenance and peer review Not commissioned; externally peer reviewed.