| Controls(n = 130) | ACL(n = 117) | ACL subgroups, based on mechanism of injury | |||
| Direct(n = 15) | Indirect(n = 18) | Non-contact(n = 50) | |||
| GG genotype, % | 70.0 (n = 91) | 68.4 (n = 80) | 80.0 (n = 12) | 66.7 (n = 12) | 72.0 (n = 36) |
| GT genotype, % | 25.4 (n = 33) | 31.6 (n = 37) | 20.0 (n = 3) | 33.3 (n = 6) | 28.0 (n = 14) |
| TT genotype, %* | 4.6 (n = 6) | 0.0 (n = 0) | 0.0 (n = 0) | 0.0 (n = 0) | 0.0 (n = 0) |
| G allele, % | 82.7 (n = 215) | 84.2 (n = 197) | 90.0 (n = 27) | 83.3 (n = 30) | 86.0 (n = 86) |
| T allele, % | 17.3 (n = 45) | 15.8 (n = 37) | 10.0 (n = 3) | 16.7 (n = 6) | 14.0 (n = 14) |
ACL, anterior cruciate ligament.
*Owing to the absence of participants with a TT genotype in the ACL group and three subgroups, the GT and TT participants were combined and compared with the GG participants.
Controls vs ACL genotypes, p = 0.890.
Controls vs ACL, TT genotype vs GG + GT genotypes, p = 0.031, OR = 0.08, 95% CI <0.01 to 1.46.
Controls vs non-contact genotypes, p = 0.935.
Controls vs ACL alleles, p = 0.745.
Controls vs non-contact alleles, p = 0.548.
Data are % (n).