Epileptic seizure activity in the acute phase following cortical impact trauma in rat
Reference (35)
- et al.
Seizure related elevations of extracellular amino acids in the human epileptic focus
Neurosci. Lett.
(1992) Glutamate toxicity and diseases of the central nervous system
Neuron
(1988)- et al.
Responses to cortical injury. I. Methodology and local effects of contusions in the rat
Brain Res.
(1981) - et al.
Brain concussion in the rat
Exp. Neurol.
(1972) - et al.
Extracellular amino acid levels in hippocampus during pilocarpine-induced seizures
Epilepsy Res.
(1993) - et al.
Spectral analysis of the electroencephalographic response to experimental concussion in the rat
Electroencephalogr. Clin. Neurophysiol.
(1982) - et al.
Thresholds in cerebral ischemia — the ischemic penumbra
Stroke
(1981) - et al.
Diagnosis and treatment of head injury in adults
The search for the neuronal mechanisms in epilepsy: an overview
Neurology
(1974)- et al.
Continuous measurement of regional blood flow in traumatic brain injury by laser doppler flowmetry
J. Neurotrauma
(1992)
Excitatory amino acid antagonists and epilepsy
Biochem. Soc. Trans.
(1993)
New wave of research in the epilepsies
The role of excitatory amino acid and NMDA receptors in traumatic brain injury
Science
(1989)
Pathology and pathophysiology of head injury
Ishaemic brain damage is still common in fatal non-missile head injury
J. Neurol. Neurosurg. Psychiat.
(1989)
Neurotransmitter-mediated mechanisms of traumatic brain injury: acetylcholine and excitatory amino acids
J. Neurotrauma
(1992)
Dynamics of extracellular metabolites in the striatum after middle cerebral artery occlusion in the rat monitored by intracerebral microdialysis
J. Cereb. Blood Flow Metab.
(1989)
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2019, Neurobiology of DiseaseCitation Excerpt :It has been suggested that LFPI produces more severe cortical damage and epileptogenesis than midline FPI (D'Ambrosio et al., 2004). The CCI model uses a piston to directly impact the brain, again through a craniotomy with the dura intact, causing tissue damage that includes crushing, laceration, cortical contusion, compression contusion, and hemorrhage (Bolkvadze and Pitkanen, 2012; Bolkvadze et al., 2015; Nilsson et al., 1994; Smith, 2016). One advantage of CCI over FPI is greater control of the impact parameters, e.g. velocity, dwell time, shape, and size of the piston allowing for a more uniform injury model (Lighthall et al., 1989).
Rat models of central nervous system injury
2019, The Laboratory RatAnimal models of post-traumatic epilepsy
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