Marijuana's interaction with brain reward systems: Update 1991
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2019, Inorganic Chemistry CommunicationsCitation Excerpt :Δ9-Tetrahydrocannabinol (THC) is the principal psychoactive ingredient of marijuana, expressing its pharmacological effects by G protein-coupled central cannabinoid receptors in the brain [1,2]. In addition, it has been proven that THC could indirectly interact with μ and δ opioid receptors and facilitates brain reward circuitry in the nucleus accumbens and ventral tegmental area [3]. As a result of complex interactions with many biological systems, the illicit use of marijuana leads to various negative health effects and increase risks of many diseases e. g. infectious diseases, pulmonary diseases, cardiovascular diseases, neurologic disease [4].
β-lactams modulate astroglial glutamate transporters and attenuate dependence to CP 55,940, a CB1 receptor agonist, in rat model
2019, Behavioural Brain ResearchThe Cannabinoid System in Nicotine Dependence and Withdrawal
2017, Negative Affective States and Cognitive Impairments in Nicotine DependenceClozapine and SCH 23390 prevent the spatial working memory disruption induced by Δ<sup>9</sup>-THC administration into the medial prefrontal cortex
2011, Brain ResearchCitation Excerpt :It has long been appreciated that dopamine (DA) has a powerful influence on the cognitive functions of the PFC, including WM (Brozoski et al., 1979; Sawaguchi and Goldman-Rakic, 1991; Goldman-Rakic, 1996; Zahrt et al, 1997; Lidow et al, 2003; Robbins and Arnsten, 2009). Additionally, interactions between DA release and cannabinoids have been reported in several brain areas in vitro and in vivo (Gardner and Lowinson, 1991; Fernández-Ruiz et al, 2010). These interactions consist in enhancement of DA release induced by cannabinoids (Poddar and Dewey, 1980; Jentsch et al., 1998; Bossong et al., 2009), no effect of cannabinoids over dopaminergic neurons (Szabo et al., 1999), and inhibition of DA release (Cadogan et al., 1997).
Neurobiological mechanisms of cannabinoid addiction
2008, Molecular and Cellular Endocrinology